Introduction
Aquaporins (AQPs) are a family of channel
proteins that allow water or very small solutes to pass,
functioning in tissues where the rapid and regulated transport
of fluid is necessary, such as the kidney, lung, and salivary
glands.
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one of the group of cell membrane
transporting proteins.
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a water channel protein expressed on the
apical surface of alveolar epithelial type I cells in distal rat
lung, suggesting a role for AQP5 in regulating alveolar surface
liquid tonicity and/or cell volume.
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an apical plasma membrane (APM) water channel
in salivary glands, lacrimal glands, and airway epithelium, has
an important role in fluid secretion.
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a water channel protein and is considered to
play an important role in water movement across the plasma
membrane.
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Normal Expression
a water channel protein expressed on the
apical surface of alveolar epithelial type I cells in distal rat
lung.
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a water-selective channel protein that is
expressed in lacrimal glands, salivary glands, and distal lung.
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Water channel aquaporin 5 (AQP5) is present
in the apical membrane of the salivary gland acinar cells.
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AQP5 was strongly labeled in alveolar
epithelial type I cells, and was also expressed in the secretory
epithelium plasma membrane in the airway.
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expressed in epithelia of lung, cornea, and
various secretory glands, sites where extracellular osmolality
is known to fluctuate.
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AQP5 is localized in the apical membranes of
several different epithelia in vivo, and in stably transfected
MDCK-II cells grown as a polarized monolayer.
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AQP5 was detected in the secretory granule
membranes by immunoblot analysis. The immunoelectron microscopy
experiments confirmed that AQP5 was to be found in the secretory
granule membrane.
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AQP5 expressed mainly in the membrane and
cytoplasm of the epithelium in the glands, ducts and cilia.
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Aquaporin-5 was found in the apical domain of
the acinar cell plasma membrane in healthy BALB/c mice.
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AQP5 is localized mainly in the apical
membrane, including intercellular secretory canaliculi of
secretory cells in the minor salivary glands, pyloric glands,
and duodenal glands.
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AQP5 displayed specific cochlear expression,
first detectable at E15.5 in the nonsensory epithelium and later
restricted to the lateral wall of the cochlear duct near the
spiral prominence. AQP5 expression continued through postnatal
periods with a change of expression domain to the stria
vascularis between postnatal day 7 (P7) and P14.
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Immunolocalization with specific antibodies
revealed strong expression of AQP5 at the luminal membrane of
secretory epithelial cells in sweat glands in mouse paw skin.
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skeletal muscle cells express AQP5 protein
and its expression is regulated by differentiation and
hypertonic stress.
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AQP5 was found in secretory acinar cells of
submandibular, parotid, and sublingual glands, where it was
restricted to apical membranes including intercellular secretory
canaliculi. In the submandibular gland, abundant AQP5 was also
found additionally at the apical membrane of intercalated duct
cells. Upon stimulation by isoproterenol, apical staining for
AQP5 in parotid acinar cells tended to appear as clusters of
dots.
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Abnormal Expression
location of AQP5 in hyperoxia groups was not
changed, but the expression of AQP5 mRNA had a notable gradual
decline when the time of hyperoxia exposure was prolonged,
compared to control group (all P<0.05).
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expression of AQP5 in the experimental groups
were reduced compared with that in the Air control group 3 days
after hyperoxia-exposure and the decrease of AQP5 expression was
associated with duration of hyperoxia-exposure.
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level of AQP-5 expression was substantially
reduced in animals with long-term anhidrosis, hence implicating
water channel impairment as a possible factor in the development
of this disorder.
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aquaporin-5 was found in the apical and
basolateral acinar plasma cell membrane in parotid,
submandibular, and sublingual glands in NOD model of Sjogren's
syndrome mice.
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Abnormal distribution of AQP5 in salivary
gland acini is likely to contribute to the deficiency of fluid
secretion, which is a defining feature of Sjogren's syndrome.
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Expression Alteration
Although several AQP genes are expressed in
the uterus, only direct activation of AQP5 could be detected
following treatment with estrogen.
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In the basal state, AQP5 was localized mainly
in the apical membrane of the acinar cell. It was also present
in the basolateral membrane to a lesser extent. When
isoproterenol (IPR) was administered to mice, dot-like,
vesicle-like and vacuole-like labeling for AQP5 was seen in the
subapical regions by light microscopy. By immunoelectron
microscopy, AQP5 was localized at both the apical and
basolateral plasma membranes in the basal state. At 5 and 30 min
after the IPR-administration, acinar lumen became enlarged and
small invaginations formed by fusion of secretory granules were
seen. AQP5 was positive along the apical plasma membrane and its
small invaginations. At 60 min, large invaginations of the lumen
were formed. AQP5 remained positive in the membrane of these
large invaginations. At 6 h, large invaginations disappeared and
AQP5 was localized in the apical plasma membrane. AQP5 was
restricted to plasma membranes and continuous invaginations
formed by the exocytosis of secretory granules. AQP5 was not
detected in the cytoplasm.
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expression of aquaporin-5, the major water
channel expressed in alveolar, tracheal, and upper bronchial
epithelium, is significantly down-regulated during acute lung
injury.
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Function
AQP5 plays an important role in maintaining
water homeostasis in the lung.
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hyperosmotic stress is an important activator
of AQP-5 in human airway epithelium, leading to significantly
increased transepithelial water permeability.
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AQP5 is responsible for the majority of water
transport across the apical membrane of type I alveolar
epithelial cells. The unimpaired alveolar fluid clearance in
AQP5-null mice indicates that high alveolar water permeability
is not required for active, near-isosmolar fluid transport.
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restricted expression of AQP5 to the apical turns of the cochlea suggests its potential role in low frequency hearing. PMID: 10527857
AQP5 is one of the candidate molecules
responsible for the water movement in the salivary glands.
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Applications
Electron Microscopy (EM)
We performed immunoperoxidase,
immunofluorescence and immunoelectron microscopy to examine
changes of AQP5-distribution during the fusion process of
secretory granule membranes into the apical membrane and
subsequent recovery process in the mouse parotid gland by
administering isoproterenol (IPR) in vivo.
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AQP5 was detected in the secretory granule
membranes by immunoblot analysis. The immunoelectron microscopy
experiments confirmed that AQP5 was to be found in the secretory
granule membrane.
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Immunofluorescence (IF)
Expression of aquaporin-5 (AQP5), studied by immunofluorescence and confocal microscopy, showed an increase in parallel with the increase in P ( f ) following hyperosmotic stress. The AQP5 was localized both in cytoplasmic vesicles and in apical cell membranes. PMID: 17043812
Localizations of AQP5 and MUC5AC in
SPC-A1cells were detected by Immunofluorescence.
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distribution of AQP5 in normal rat nasal
mucosa and nasal mucosa in rat with allergic rhinitis were
observed by immunofluorescence technique.
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Aquaporin-5 was stained using avidin-biotin-peroxidase
complex and indirect immunofluorescence staining methods, and
visualized using light and laser scanning confocal microscopy.
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Immunofluorescence and Western blotting
strongly suggested that LPS altered AQP5 subcellular
distribution from an intracellular vesicular compartment to the
plasma membrane.
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We used indirect immunofluorescence and an
immunoperoxidase technique to assess expression and subcellular
localisation of aquaporin 5 in patients and controls.
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Immunohistochemistry (IHC)
expression of AQP5 mRNA level and the location were detected by reverse transcription-polymerase chain reaction and immunohistochemistry respectively. PMID: 16887054
Immunohistochemistry revealed a strong
AQP-5-like activity reaction at the apical membrane of the
glandular secretory cells, which was absent from the surrounding
myoepithelium and all other skin structures.
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expression of AQP5 protein and mRNA in 65
cases epithelial ovarian tumors and 13 cases normal tissue were
measured by immunohistochemical technique, Western blotting and
RT-PCR, respectively.
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Furthermore, the expression of AQP5 in normal
rat nasal mucosa and nasal mucosa from rat with allergic
rhinitis were studied by immunohistochemical staining.
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expression of aquaporin-5 was investigated by
immunohistochemical studies and by polymerase chain reaction.
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We surveyed the expression and
immunohistochemical localization of AQP5 in the rat digestive
system.
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Immunohistochemical analysis of the SMG
demonstrated that the AQP5 protein was localized in the basal
and apical/lateral plasma membrane of acinar cells in rats
expressing the high level of AQP5. In the rat expressing the low
level, however, this channel protein was localized strongly in
the apical/lateral plasma membrane, but only very weakly in the
basal membrane of the acinar cells. Such a diverse localization
of AQP5 was confirmed by Western blotting as well.
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RT-PCR, immunohistochemistry and Western blot
analysis revealed that C2C12 cells express AQP5.
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Western Blotting (WB)
expressions of AQP1 and AQP5 were examined by
Western Blot in the lung tissue under hyperoxia.
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Western blot analysis showed that sweat gland
cells from freely sweating horses expressed the water channel
aquaporin-5 (AQP-5).
PMID: 16412116
expression of AQP5 protein and mRNA in 65
cases epithelial ovarian tumors and 13 cases normal tissue were
measured by immunohistochemical technique, Western blotting and
RT-PCR, respectively.
PMID: 16242760
AQP5 was detected in the secretory granule
membranes by immunoblot analysis. The immunoelectron microscopy
experiments confirmed that AQP5 was to be found in the secretory
granule membrane.
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We analyzed the change in expression of
inducible NOS (iNOS), neuronal NOS (nNOS), endothelial NOS (eNOS),
aquaporin 1 (AQP1) and aquaporin 5 (AQP5) over time by Western
blot.
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By Western blot analysis, the expression
level of aquaporin (AQP) 5 in the submandibular gland (SMG) was
found to be different among individual rats of the Sprague-Dawley
(SD) strain.
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Reverse transcription-polymerase chain
reaction and immunoblot analyses revealed that mRNAs encoding
AQP1, 4 and 5 (but not 2 or 3) subtypes were expressed in rat
middle ear epithelium.
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Immunoblot of cochlear homogenate yielded a
predominant AQP5-immunoreactive band of M(r) 35 kDa.
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